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Tirzepatide
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Tirzepatide 10mg

Longevity

$169

4.0 (24 reviews)

Dual GLP-1/GIP receptor agonist for metabolic optimization, appetite regulation, and body composition improvement.

Available in 10mg vial, lyophilized powder
Dual agonist: GLP-1 and GIP receptors
Third-party tested for purity and potency
Requires reconstitution with bacteriostatic water
1
Third-party tested

Overview

Tirzepatide is a first-in-class dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist. FDA-approved as Mounjaro for type 2 diabetes and Zepbound for obesity, tirzepatide demonstrated up to 22.5% weight reduction in the SURMOUNT-1 trial. Its dual mechanism provides complementary metabolic benefits that surpass GLP-1 agonism alone.

Lab Results — 10mg

Every batch is independently tested. Results below are from the Certificate of Analysis for the 10mg batch.

Test
Result
Status
Identity (HPLC-MS)
Confirmed Tirzepatide
Pass
Purity
99.2%
Pass
Heavy Metals (Pb)
< 0.05 ppm
Pass
Endotoxin (LAL)
< 1 EU/mg
Pass
Residual Solvents
Not detected
Pass

Scientific Details

Molecular Formula

C₂₂₅H₃₄₈N₄₈O₆₈

Molecular Weight

~4.8 kDa

Form

Lyophilized peptide powder

Solubility

Soluble in bacteriostatic water

Dosage Range

2.5–15mg weekly (research context)

Plasma Half-Life

~5 days

Bioavailability

~80% subcutaneous

Resources

Key peer-reviewed studies supporting the efficacy and safety of this compound.

Tirzepatide once weekly for the treatment of obesity (SURMOUNT-1)

Jastreboff A.M. et al.New England Journal of Medicine (2022)

Key FindingTirzepatide produced a mean weight reduction of up to 22.5% over 72 weeks in adults with obesity, significantly exceeding placebo.

Dual GIP and GLP-1 receptor agonist tirzepatide improves glycemic control

Frias J.P. et al.Lancet (2021)

Key FindingTirzepatide demonstrated superior HbA1c reduction and weight loss compared to semaglutide in a head-to-head Phase 3 trial.

GIP receptor agonism attenuates GLP-1 receptor agonist-induced nausea

Samms R.J. et al.Diabetes (2020)

Key FindingCo-activation of GIP receptors reduced the nausea side-effect profile of GLP-1 agonism while preserving or enhancing metabolic benefits.